Neurocognitive DisordersHealthy VolunteersMDMA

Effects of a β-blocker on the cardiovascular response to MDMA (Ecstasy)

This double-blind cross-over study (n=16) finds that pindolol (medication for hypertension, 20mg) prevents the heart-raising effects of MDMA (112mg/70kg) but not the elevation of blood pressure.

Authors

  • Matthias Liechti
  • Franz Vollenweider
  • Cédric Hysek

Published

BMJ Open
individual Study

Abstract

Background

MDMA (3,4-methylenedioxymethamphetamine, ‘Ecstasy’) produces tachycardia and hypertension and is rarely associated with cardiovascular and cerebrovascular complications. In clinical practice, β-blockers are often withheld in patients with stimulant intoxication because they may increase hypertension and coronary artery vasospasm due to loss of β2-mediated vasodilation and unopposed α-receptor activation. However, it is unknown whether β-blockers affect the cardiovascular response to MDMA.

Methods

The effects of the non-selective β-blocker pindolol (20 mg) on the cardiovascular effects of MDMA (1.6 mg/kg) were investigated in a double-blind placebo-controlled crossover study in 16 healthy subjects.

Results

Pindolol prevented MDMA-induced increases in heart rate. Peak values (mean±SD) for heart rate were 84±13 beats/min after MDMA vs 69±7 beats/min after pindolol-MDMA. In contrast, pindolol pretreatment had no effect on increases in mean arterial blood pressure (MAP) after MDMA. Peak MAP values were 115±11 mm Hg after MDMA vs 114±11 mm Hg after pindolol-MDMA. Pindolol did not change adverse effects of MDMA.

Conclusion

The results of this study indicate that β-blockers may prevent increases in heart rate but not hypertensive and adverse effects of MDMA.

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Research Summary of 'Effects of a β-blocker on the cardiovascular response to MDMA (Ecstasy)'

Introduction

Earlier research documents that MDMA (3,4-methylenedioxymethamphetamine, 'Ecstasy') produces marked cardiostimulant effects — notably tachycardia and hypertension — and has been rarely associated with serious vascular events such as myocardial infarction, intracranial haemorrhage and cerebral infarction. In clinical practice, the use of β-adrenergic blockers (β-blockers) in stimulant intoxication is controversial because blockade of β-receptors may, theoretically, remove β 2 -mediated vasodilation and leave unopposed α-receptor vasoconstriction, thereby worsening hypertension or coronary vasospasm. It was unknown whether β-blockers alter the cardiovascular responses to MDMA in humans. M. and colleagues set out to assess the haemodynamic effects of the non-selective β-blocker pindolol on MDMA-induced cardiovascular changes in healthy volunteers. The study aimed to test whether pindolol modifies MDMA-induced increases in heart rate, blood pressure and body temperature, and whether it alters acute adverse effect burden. The cardiovascular and adverse-effect outcomes reported here were secondary outcomes from a controlled human study whose primary subjective and neurocognitive results were reported elsewhere.

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Study Details

References (3)

Papers cited by this study that are also in Blossom

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Hasler, F., Ludewig, S. · Journal of Psychopharmacology (2008)

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