Healthy VolunteersChronic PainHeadache Disorders (Cluster & Migraine)Safety & Risk ManagementPsilocybin

Psilocybin dose-dependently causes delayed, transient headaches in healthy volunteers

This double-blind placebo-controlled study (n=18) found that psilocybin (5-30mg/70kg) frequently caused mild to moderate delayed and transient headaches in healthy volunteers in a dose-dependent manner.

Authors

  • Roland Griffiths
  • Matthew Johnson
  • Andrew Sewell

Published

Drug and Alcohol Dependence
individual Study

Abstract

Background

Psilocybin is a well-characterized classic hallucinogen (psychedelic) with a long history of religious use by indigenous cultures, and nonmedical use in modern societies. Although psilocybin is structurally related to migraine medications, and case studies suggest that psilocybin may be efficacious in treatment of cluster headache, little is known about the relationship between psilocybin and headache.

Methods

This double-blind study examined a broad range of psilocybin doses (0, 5, 10, 20, and 30. mg/70. kg) on headache in 18 healthy participants.

Results

Psilocybin frequently caused headache, the incidence, duration, and severity of which increased in a dose-dependent manner. All headaches had delayed onset, were transient, and lasted no more than a day after psilocybin administration.

Conclusions

Possible mechanisms for these observations are discussed, and include induction of delayed headache through nitric oxide release. These data suggest that headache is an adverse event to be expected with the nonmedical use of psilocybin-containing mushrooms as well as the administration of psilocybin in human research. Headaches were neither severe nor disabling, and should not present a barrier to future psilocybin research.

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Research Summary of 'Psilocybin dose-dependently causes delayed, transient headaches in healthy volunteers'

Introduction

Classic hallucinogens such as psilocybin produce psychoactive effects largely via 5-HT2A receptor agonism and have a long history of ritual and recreational use. Earlier clinical reports and case series have intermittently noted headache following administration of classic psychedelics, and some small studies and reviews suggested a dose-related occurrence of head pain after psilocybin, but these observations were generally retrospective, inconsistent, or not systematically characterised. At the same time, several tryptamine-based and indole compounds are used in acute and prophylactic headache management, and anecdotal reports and case series have raised the possibility that psilocybin or related compounds might both provoke and, paradoxically, treat certain primary headache syndromes such as cluster headache. Johnson and colleagues designed the present study to prospectively and systematically characterise headache as an adverse event after controlled psilocybin administration. The investigators aimed to examine whether headache incidence, timing, duration and severity were dose-dependent across a broad range of oral psilocybin doses in medically and psychiatrically healthy volunteers, and to explore relationships between headache and other measured drug effects and physiological responses.

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Study Details

References (12)

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